![Pathogens | Free Full-Text | Tissue Pathogens and Cancers: A Review of Commonly Seen Manifestations in Histo- and Cytopathology Pathogens | Free Full-Text | Tissue Pathogens and Cancers: A Review of Commonly Seen Manifestations in Histo- and Cytopathology](https://www.mdpi.com/pathogens/pathogens-10-01410/article_deploy/html/images/pathogens-10-01410-g001.png)
Pathogens | Free Full-Text | Tissue Pathogens and Cancers: A Review of Commonly Seen Manifestations in Histo- and Cytopathology
![Frontiers | UFL1, a UFMylation E3 ligase, plays a crucial role in multiple cellular stress responses Frontiers | UFL1, a UFMylation E3 ligase, plays a crucial role in multiple cellular stress responses](https://www.frontiersin.org/files/Articles/1123124/fendo-14-1123124-HTML/image_m/fendo-14-1123124-g001.jpg)
Frontiers | UFL1, a UFMylation E3 ligase, plays a crucial role in multiple cellular stress responses
![Smc5/6 functions with Sgs1-Top3-Rmi1 to complete chromosome replication at natural pause sites | Nature Communications Smc5/6 functions with Sgs1-Top3-Rmi1 to complete chromosome replication at natural pause sites | Nature Communications](https://media.springernature.com/full/springer-static/image/art%3A10.1038%2Fs41467-021-22217-w/MediaObjects/41467_2021_22217_Fig1_HTML.png)
Smc5/6 functions with Sgs1-Top3-Rmi1 to complete chromosome replication at natural pause sites | Nature Communications
![DNA end resection and its role in DNA replication and DSB repair choice in mammalian cells | Experimental & Molecular Medicine DNA end resection and its role in DNA replication and DSB repair choice in mammalian cells | Experimental & Molecular Medicine](https://media.springernature.com/full/springer-static/image/art%3A10.1038%2Fs12276-020-00519-1/MediaObjects/12276_2020_519_Fig1_HTML.png)
DNA end resection and its role in DNA replication and DSB repair choice in mammalian cells | Experimental & Molecular Medicine
![Frontiers | Rad5 participates in lesion bypass through its Rev1-binding and ubiquitin ligase domains, but not through its helicase function Frontiers | Rad5 participates in lesion bypass through its Rev1-binding and ubiquitin ligase domains, but not through its helicase function](https://www.frontiersin.org/files/Articles/1062027/fmolb-09-1062027-HTML/image_m/fmolb-09-1062027-g001.jpg)
Frontiers | Rad5 participates in lesion bypass through its Rev1-binding and ubiquitin ligase domains, but not through its helicase function
![The multifaceted roles of PARP1 in DNA repair and chromatin remodelling | Nature Reviews Molecular Cell Biology The multifaceted roles of PARP1 in DNA repair and chromatin remodelling | Nature Reviews Molecular Cell Biology](https://media.springernature.com/full/springer-static/image/art%3A10.1038%2Fnrm.2017.53/MediaObjects/41580_2017_Article_BFnrm201753_Fig1_HTML.jpg)
The multifaceted roles of PARP1 in DNA repair and chromatin remodelling | Nature Reviews Molecular Cell Biology
![Frontiers | The Replication Stress Response on a Narrow Path Between Genomic Instability and Inflammation Frontiers | The Replication Stress Response on a Narrow Path Between Genomic Instability and Inflammation](https://www.frontiersin.org/files/Articles/702584/fcell-09-702584-HTML-r1/image_m/fcell-09-702584-g001.jpg)
Frontiers | The Replication Stress Response on a Narrow Path Between Genomic Instability and Inflammation
![JoF | Free Full-Text | Post-Translational Modifications of PCNA: Guiding for the Best DNA Damage Tolerance Choice JoF | Free Full-Text | Post-Translational Modifications of PCNA: Guiding for the Best DNA Damage Tolerance Choice](https://pub.mdpi-res.com/jof/jof-08-00621/article_deploy/html/images/jof-08-00621-g001.png?1654850522)
JoF | Free Full-Text | Post-Translational Modifications of PCNA: Guiding for the Best DNA Damage Tolerance Choice
![RIF1 promotes replication fork protection and efficient restart to maintain genome stability | Nature Communications RIF1 promotes replication fork protection and efficient restart to maintain genome stability | Nature Communications](https://media.springernature.com/m685/springer-static/image/art%3A10.1038%2Fs41467-019-11246-1/MediaObjects/41467_2019_11246_Fig1_HTML.png)
RIF1 promotes replication fork protection and efficient restart to maintain genome stability | Nature Communications
![Eukaryotic clamp loaders and unloaders in the maintenance of genome stability | Experimental & Molecular Medicine Eukaryotic clamp loaders and unloaders in the maintenance of genome stability | Experimental & Molecular Medicine](https://media.springernature.com/full/springer-static/image/art%3A10.1038%2Fs12276-020-00533-3/MediaObjects/12276_2020_533_Fig1_HTML.png)
Eukaryotic clamp loaders and unloaders in the maintenance of genome stability | Experimental & Molecular Medicine
![Genes | Free Full-Text | Control of Eukaryotic DNA Replication Initiation—Mechanisms to Ensure Smooth Transitions Genes | Free Full-Text | Control of Eukaryotic DNA Replication Initiation—Mechanisms to Ensure Smooth Transitions](https://pub.mdpi-res.com/genes/genes-10-00099/article_deploy/html/images/genes-10-00099-g001.png?1571016814)
Genes | Free Full-Text | Control of Eukaryotic DNA Replication Initiation—Mechanisms to Ensure Smooth Transitions
![DNA double-strand break repair-pathway choice in somatic mammalian cells | Nature Reviews Molecular Cell Biology DNA double-strand break repair-pathway choice in somatic mammalian cells | Nature Reviews Molecular Cell Biology](https://media.springernature.com/full/springer-static/image/art%3A10.1038%2Fs41580-019-0152-0/MediaObjects/41580_2019_152_Fig1_HTML.png)
DNA double-strand break repair-pathway choice in somatic mammalian cells | Nature Reviews Molecular Cell Biology
![Cells | Free Full-Text | The Interplay of Cohesin and the Replisome at Processive and Stressed DNA Replication Forks Cells | Free Full-Text | The Interplay of Cohesin and the Replisome at Processive and Stressed DNA Replication Forks](https://pub.mdpi-res.com/cells/cells-10-03455/article_deploy/html/images/cells-10-03455-g001.png?1639383300)
Cells | Free Full-Text | The Interplay of Cohesin and the Replisome at Processive and Stressed DNA Replication Forks
![Two replication fork remodeling pathways generate nuclease substrates for distinct fork protection factors | Science Advances Two replication fork remodeling pathways generate nuclease substrates for distinct fork protection factors | Science Advances](https://www.science.org/cms/10.1126/sciadv.abc3598/asset/12f4fa75-d955-4f40-b13d-48cb9b730c29/assets/graphic/abc3598-f1.jpeg)
Two replication fork remodeling pathways generate nuclease substrates for distinct fork protection factors | Science Advances
![IJMS | Free Full-Text | DNA2 in Chromosome Stability and Cell Survival—Is It All about Replication Forks? IJMS | Free Full-Text | DNA2 in Chromosome Stability and Cell Survival—Is It All about Replication Forks?](https://www.mdpi.com/ijms/ijms-22-03984/article_deploy/html/images/ijms-22-03984-g001.png)
IJMS | Free Full-Text | DNA2 in Chromosome Stability and Cell Survival—Is It All about Replication Forks?
Super-resolution visualization of distinct stalled and broken replication fork structures | PLOS Genetics
![The plasticity of DNA replication forks in response to clinically relevant genotoxic stress | Nature Reviews Molecular Cell Biology The plasticity of DNA replication forks in response to clinically relevant genotoxic stress | Nature Reviews Molecular Cell Biology](https://media.springernature.com/m685/springer-static/image/art%3A10.1038%2Fs41580-020-0257-5/MediaObjects/41580_2020_257_Fig1_HTML.png)
The plasticity of DNA replication forks in response to clinically relevant genotoxic stress | Nature Reviews Molecular Cell Biology
![53BP1 Mediates ATR-Chk1 Signaling and Protects Replication Forks under Conditions of Replication Stress | Molecular and Cellular Biology 53BP1 Mediates ATR-Chk1 Signaling and Protects Replication Forks under Conditions of Replication Stress | Molecular and Cellular Biology](https://journals.asm.org/cms/asset/ee33e114-648c-477b-89b7-7700c0500485/mcb.2018.38.issue-8.largecover.jpg)
53BP1 Mediates ATR-Chk1 Signaling and Protects Replication Forks under Conditions of Replication Stress | Molecular and Cellular Biology
![Therapy resistance: opportunities created by adaptive responses to targeted therapies in cancer | Nature Reviews Cancer Therapy resistance: opportunities created by adaptive responses to targeted therapies in cancer | Nature Reviews Cancer](https://media.springernature.com/full/springer-static/image/art%3A10.1038%2Fs41568-022-00454-5/MediaObjects/41568_2022_454_Fig1_HTML.png)